In this paper, the network transmission properties of a feedforward Spiking Neural Network (SNN) affected by synchronous stimuli are investigated with respect to the connection probability and the synaptic strengths. By means of an event-driven method, all simulations are conducted using the Leaky Integrate-and-Fire with Latency (LIFL) model. Typical cases are taken into consideration, in which a network section (module) is able to process the input information, introducing a particular behavior, that we have called path multimodality. Simulation results are discussed. Through this phenomenon, the output layer of the network can generate a number of temporally spaced groups of synchronous spikes. The multimodality effect could be applied for various purposes, for instance in coding or else transmission issues.
[Vydáno k výstavě pořádané Správou Pražského hradu a Židovským muzeem v Praze k 400. výročí úmrtí Rabiho Jehudy Levy ben Becalela, Císařská konírna, 5. 8. - 8. 11. 2009.]
At the analysis of clinical efficiency of differential therapy in the patients with AP with BO the type vegetative nervous system is defined. In the patients with hypersympathicotonic VR (group II) and asympathycotonic VR (III group) during differential treatment there was noted reduction in duration of dyspnea, pulmonary cyanosis, moist pulmonary rales . In the patients with AP with BO alongside with changes biochemical, immune and vegetative status there was found wrong attitude of the family to the child, and also the psychological behavioral deviations in the child as increased anxiety, aggression; on a background of complex therapy were performed psychological correctional methods of treatment: musictherapy, psychogymnastics, game exercises. At catamnestic observation within one year of 40 children who have received on a background of other methods of treatment psychocorrection, repeated bronchial obstruction was observed only at 15 % observable children. In control group already through 3 months 45 % of children have addressed with repeated episodes BO at pneumonia and bronchitis., M. M. Khaidarova, and Literatura
Experimental studies in animals provide relevant knowledge about pathogenesis of radiation-induced injury to the central nervous system. Radiation-induced injury can alter neuronal, glial cell population, brain vasculature and may lead to molecular, cellular and functional consequences. Regarding to its fundamental role in the formation of new memories, spatial navigation and adult neurogenesis, the majority of studies have focused on the hippocampus. Most recent findings in cranial radiotherapy revealed that hippocampal avoidance prevents radiation-induced cognitive impairment of patients with brain primary tumors and metastases. However, numerous preclinical studies have shown that this problem is more complex. Regarding the fact, that the radiation-induced cognitive impairment reflects hippocampal and non-hippocampal compartments, it is highly important to investigate molecular, cellular and functional changes in different brain regions and their integration at clinically relevant doses and schedules. Here, we provide a literature review in order support the translation of preclinical findings to clinical practice and improve the physical and mental status of patients with brain tumors.