Obsahuje přívazek: Spolky na vzájemné pomáhání / sepsal F. S. Kodym (Vyňato z Hospodářských Novin 1860) ; V Praze a ve Vídni : Kober & Markgraf, 1860 ; 49 stran, Obsahuje přívazek: K theorii statistiky průmyslu / sepsal Josef Erben (Řeč úvodní, učiněná dne 12. listopadu 1862 při počátku čtení o statistice průmyslu na témž ústavě) ; V Praze : Nákladem kněhkupectví: I. L. Kober, 1863 ; 15 stran, Obsahuje přívazek: Centrální trhovna na potravu ve Vídní, c. k. hlavním a sídelním městě říšském ; Ce Vídni : Ve skladu správy trhovny na potravu, 1865 ; 15 stran, and Obsahuje přívazek: Central-Markthalle der k. k. Reichshaupt- und Residenzstadt Wien ; Wien : Verlag der Hallen-Verwaltung, 1865 ; 15 stran
Václav Rubeška., (Předneseno v lidových přednáškách c.k. České university v Praze v kursu 1916, pořádaném péčí České eugenické společnosti v Praze), and Přívazek k : Jaroslav Kříženecký / Ochrana mládeže a eugenika
Associations between different infectious agents and obesity have been reported in humans for over thirty years. In many cases, as in nosocomial infections, this relationship reflects the greater susceptibility of obese individuals to infection due to impaired immunity. In such cases, the infection is not related to obesity as a causal factor but represents a complication of obesity. In contrast, several infections have been suggested as potential causal factors in human obesity. However, evidence of a causal linkage to human obesity has only been provided for adenovirus 36 (Adv36). This virus activates lipogenic and proinflammatory pathways in adipose tissue, improves insulin sensitivity, lipid profile and hepatic steatosis. The E4orf1 gene of Adv36 exerts insulin senzitizing effects, but is devoid of its pro-inflammatory modalities. The development of a vaccine to prevent Adv36- induced obesity or the use of E4orf1 as a ligand for novel antidiabetic drugs could open new horizons in the prophylaxis and treatment of obesity and diabetes. More experimental and clinical studies are needed to elucidate the mutual relations between infection and obesity, identify additional infectious agents causing human obesity, as well as define the conditions that predispose obese individuals to specific infections., V. Hainer, H. Zamrazilová, M. Kunešová, B. Bendlová, I. Aldhoon-Hainerová., and Obsahuje bibliografii
The functional antagonism between obestatin and ghrelin in the testis is under investigation. We investigated the ability of obestatin to counteract the inhibitory effect of ghrelin on basal and stimulated testosterone (T) secretion in vitro. Testicular strips from adult rats were incubated with 10 ng/ml and 100 ng /ml of obestatin alone, ghrelin alone and obestatin + ghrelin. Obestatin modulation of stimulated T secretion was evaluated by incubation of testicular samples with 10 ng/ml and 100 ng/ml obestatin, ghrelin and obestatin + ghrelin in the absence and presen ce of 10 IU of human chorionic gonadotrophin (hCG). T concentrations in the hCG treated groups were significantly (P<0.0001) higher than those in the control groups. Obestatin caused a significant increase in basal T secretion in a dose-dependent manner; however, obestatin at the both 10 ng /ml and 100 ng/ml significantly (P<0.0001) in creased hCG-stimulated T secretion. In contrast, ghrelin in a dose-dependent manner significantly (P<0.001) decreased both basal and hCG-induced T secretion by testicular slices. Obestatin opposed the inhibitory effect of ghrelin on T secretion under both basal and hCG-stimulated conditions at all doses tested. In conclusions, administration of obestatin was able to antagonize the inhibitory effect of ghrelin on testosterone secretion in vitro ., T. Afsar, S. Jahan, S. Razak, A. Almajwal, M. Abulmeaty, H. Wazir, A. Majeed., and Obsahuje bibliografii